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Modulation of Apoptotic Signaling by the Hepatitis B Virus X Protein

H 2 S oxidation by nanodisc-embedded human sulfide quinone oxidoreductase

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However, Galloway and McLellan found that while an AP-1 site at −340 of human GCLM modulated basal expression of GCLM, neither the AP-1 site nor the ARE/EpRE site at −302 was involved in mediating the up-regulation of GCLM by TBH (Galloway et al., 1998)