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In conclusion, TMAO likely modulates immune responses in IBD through the activation of the PERK-CHOP and PERK-eIF2α pathways, underscoring the complex interplay of UPR signaling in maintaining intestinal homeostasis and immune function

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Recently, a small phase I/IIa study of (in)GSH in PD demonstrated a mild symptomatic improvement in PD symptoms as measured by the Unified PD Rating Scale, with return of symptoms upon withdrawal of (in)GSH